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HEPARIN-INDUCED THROMBOCYTOPENIA IN HEMODIALYSIS. CASE REPORT AND REVIEW OF THE LITERATURE
Trombocitopenia inducida por heparina en hemodiálisis. Presentación de un caso y revisión de la literatura
F. Henríquez, A. Rodríguez, C. Mora, E. Sánchez-Deig, M. Macía, J. Navarro.
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    "idiomaDefecto" => true
    "titulo" => "HEPARIN-INDUCED THROMBOCYTOPENIA IN HEMODIALYSIS. CASE REPORT AND REVIEW OF THE LITERATURE"
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      0 => array:2 [
        "autoresLista" => "F. Henríquez, A. Rodríguez, C. Mora, E. Sánchez-Deig, M. Macía, J. Navarro."
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          0 => array:1 [
            "nombre" => "F. Henríquez, A. Rodríguez, C. Mora, E. Sánchez-Deig, M. Macía, J. Navarro."
          ]
        ]
      ]
    ]
    "titulosAlternativos" => array:1 [
      "es" => array:1 [
        "titulo" => "Trombocitopenia inducida por heparina en hemodiálisis. Presentación de un caso y revisión de la literatura"
      ]
    ]
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            0 => "hemodiálisis, heparina, trombopenia, trombosis"
          ]
        ]
      ]
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    "tieneResumen" => true
    "resumen" => array:2 [
      "en" => array:1 [
        "resumen" => "Thrombocytopenia is a potential complication of heparin therapy. There are two forms of heparin-induced thrombocytopenia (HIT). Type-I HIT is characterized by a mild decrease in platelet count that occurs within the first 2-4 days after heparin initiation. The platelet count often returns to normal without stop heparin treatment. The mechanism of thrombocytopenia appears to be due to a direct effect of heparin on platelet activation. The second form (type-II) is an immune-mediated disorder characterized by severe thrombocytopenia, which may include both arterial and venous thrombosis. We present a case of type-II HIT occurred in a hemodialysis patient resulting in acute pulmonary embolism and peripheral venous trombosis, and review the literature."
      ]
      "es" => array:1 [
        "resumen" => "La trombopenia es una complicación potencial del tratamiento con heparina. Hay dos formas de trombopenia inducida por heparina (TIH). El tipo I se caracteriza por un descenso leve de las cifras de plaquetas que ocurre de 2 a 4 días después de iniciar la terapia. El recuento plaquetario suele normalizarse sin necesidad de suspender el tratamiento. El mecanismo parece relacionarse con un efecto directo de la heparina sobre la activación plaquetaria. La segunda forma (tipo II) es un desorden de carácter inmune caracterizado por trombopenia severa, que puede asociar fenómenos trombóticos arteriales y venosos. Presentamos un caso de TIH tipo II en un paciente en hemodiálisis que resultó en tromboembolismo pulmonar agudo y trombosis venosa periférica, y hacemos una revisión de la literatura."
      ]
    ]
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  "EPUB" => "https://multimedia.elsevier.es/PublicationsMultimediaV1/item/epub/X2013251406020645?idApp=UINPBA000064"
]
Article information
ISSN: 20132514
Original language: English
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